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There were several limitations of our work, the main one being that this was an in vitro study using human cell lines in 2D cell cultures
The platform was built specifically to eliminate the guesswork and reduce the risks associated with self-directed research
3), suggesting that ALT has been activated and is responsible for the increase in telomere length seen in the cancer cells
Adding to the complexity, if the viral glycoprotein cross-seeds an endogenous brain protein, years later even the viral protein need not be present, as the run-away proteopathy would now involve only endogenous brain protein (s)
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