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The pathophysiological processes in PD, including oxidative stress, protein misfolding, mitochondrial dysfunction and neuroinflammation, overlap with those observed in type 2 diabetes (T2D) [2,3,4,5]

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Taken together these findings encourage new clinically relevant approaches to understanding the memory enhancing effects, especially of the angiotensin IV system, on cerebral blood flow, neuroprotection, stress and depression, alcohol consumption, seizure, Alzheimers and PDs, and diabetes (12, 172, 173)