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These therapeutic agents contribute to improvements in key cardiovascular risk factors, including reductions in body weight, blood pressure, and glycaemic variability, all of which are independently associated with decreased cardiovascular risk [4, 5]

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However, despite strong evidence that tirzepatide engages the GIPR in competition-binding assays, cell-based experiments using transfected receptors and studies of transgenic mice, there is little functional evidence that tirzepatide directly activates the GIPR in humans as part of its substantial pharmacological effect

The survey also asked participants whether they would restart taking a GLP-1 if they regain weight after stopping the drug