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Moreover, the PRDX2MFN2ACSL4 pathway has been implicated in mitochondria-associated ferroptosis and has been suggested to exert protective effects in the diabetic cardiac microvascular system [95].Collectively, these findings highlight that mitochondriallipid crosstalk, coordinated through lipid remodeling programs, represents a key determinant of ferroptotic sensitivity in cardiovascular pathology

[DOI] [PMC free article] [PubMed] [Google Scholar] 105.Vidal-Casariego A., Burgos-Pelez R., Martnez-Faedo C