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Eosinophilic inflammation, Th2 cytokine production, and airway reactivity were abrogated when ATP was depleted using apyrase or the P2 receptor was blocked in mice ( Extracellular ATP levels were elevated in the lungs of patients with IPF in mice with bleomycin-induced pulmonary fibrosis, in association with upregulation of P2Y2 receptor expression ( Ectonucleotidase in Chronic Pulmonary Inflammation CD39 and CD73 serve to decrease extracellular ATP levels but increase extracellular adenosine levels, thus shifting the balance from ATP signaling to adenosine signaling

It subsequently damages regions that are responsible for memory and learning over time

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