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3.1 Endothelial dysfunction Various clinical and laboratory studies have shown that endothelial dysfunction induced by cigarette smoke is mediated by decreased nitric oxide (NO) bioavailability, increased production of superoxide anions, and increased production and release of endothelin ( 28 )

Surprisingly, Latvia is 9th, with 37%, and not so surprisingly, Croatia completes the Balkan Three, with 36.9% of the population using tobacco

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In addition, TJ proteins were also significantly suppressed in lung tissue of patients with end-stage COPD and in airliquid interface differentiated epithelial cells from these patients [13]