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Chen S, Wasserman DH, MacKintosh C, Sakamoto K (2011) Mice with AS160/TBC1D4-Thr649Ala knockin mutation are glucose intolerant with reduced insulin sensitivity and altered GLUT4 trafficking

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GLP-1 secretion in response to monosaccharides and other carbohydrates The cellular mechanisms underlying glucose-stimulation of GLP-1 secretion from L-cells are, at least in part, similar to the stimulation of insulin secretion in the islets

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The acceleration in muscle mass using GLP-1s is clear

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