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31 MatsuuraH.InagakiM.MaeshigeK.IdeN.KajimuraY.ItakuraY

Importantly, the direction of causality between homocysteine and these conditions is not always clear, and more rigorous research is needed

Neuroprotective effects of GHK-Cu in models of neurodegeneration

Compared with WT SOD1, disease-linked mutant SOD1 proteins readily form monomers by reduction of the disulfide bond between Cys-57 and Cys-146 or by demetallation at the dimer interface (Tiwari and Hayward, 2003