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Because not only does all the signaling say, do something with the cholesterol, but the way you actually get cholesterol into the cell is to burn through a bunch of ATP with motor proteins, move it from outside the cell to inside the cell in order to facilitate that turnover

In conclusion, our study revealed a previously unidentified function of the ER stress sensor protein IRE1 in determining cellular sensitivity to ferroptosis through regulating GSH availability, a finding that may lead to the development of treatment strategies for ferroptosis-associated normal tissue damage and improvement in the efficacy of existing cancer therapies

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