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Description

These therapies mimic the action of the endogenous GLP-1 hormone to stimulate insulin secretion, suppress glucagon release, slow gastric emptying, and reduce appetite, making them highly effective for the management of type 2 diabetes and chronic weight management

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demonstrated that KLF15 reduced the sensitivity of VSMCs to ferroptosis by interacting with p53, thereby preventing p53-mediated repression of SLC7A11 transcription [89]

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