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PrPdeficient cerebellar neurons show impaired survival after exposure to hypoxia or treatments with glutamate or H 2 O 2 , while PrPdeficient astrocytes have enhanced GSH contents and show accelerated GSH export and reduced GT activity (Guitart et al

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Studies have analysed its involvement, for instance, in: cellular energy metabolism, mitochondrial function, processes related to the nervous system, acetyl-CoA metabolism, protecting cells from oxidative stress