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The hypercoagulopathy in patients with thalassemia has been attributed to several risk factors including abnormal RBCs, chronically activated platelets, enhanced platelet aggregation, iron overload, splenectomy, and increased prothrombin generation 6,7,8,9

TMAO and the inflammasome NLRP3 The activation of the NLRP3 inflammasome is a critical mediator of TMAO-induced pro-inflammatory effects

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