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Although GSH extrusion may provide a simple mechanism to circumvent the normally protective functions of GSH, there is increasing evidence that GSH export is required for activation of specific apoptotic signaling pathways and/or for proper dismantling of cellular components (Coppola and Ghibelli, 2000
D-Gal is a hepatocellular phosphate uracil nucleotide interference agent that is metabolized via the galactose pathway and can cause diffuse necrosis and inflammation rather than zonal necrosis, similar to most hepatotoxic drugs (153)
In vitro limiting dilution assays were performed ( j )
Dopamine precursors and brain function in phenylalanine hydroxylase deficiency
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