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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

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Zhou JN, Liu RY, Kamphorst W, Hofman MA, Swaab DF

Cats, unlike some animals, cannot produce this vitamin on their own and must obtain it through their diet, primarily from animal-based proteins like meat and fish