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The VSIG4 gene deletion reduces lactate and H3K18la production, thereby decreasing STAT3-mediated SPP1 transcription and disrupting intercellular interactions between TAMs and neutrophils, improving the immunosuppressive microenvironment (74)

While sommarutide is undoubtedly an exciting potential treatment option, there is still a lack of clear clinical data on the mechanism of action of GLP-1 agonists in NASH, such as whether GLP-1 agonists have a direct, independent role in improving NASH, or simply by improving body weight, insulin resistance, and so on

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