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ES NDs effectively reduce the abnormal accumulation of Fe 2+ , GPX4 inactivation, and lipid peroxidation in chondrocytes, thereby improving the metabolic disorders of chondrocytes caused by ferroptosis, and show significant therapeutic effects in alleviating OA

Selective compounds such as G1 (a specific agonist) and G15/G36 (specific antagonists) share a quinoline-based structure and have advanced GPER research [24,25,26]