glp 1 receptor agonists and dpp 4 inhibitors Mechanism of action for GIP, GLP-1 analogues DPP4 in Effects of incretin-based therapies on

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Electron leakage from the mitochondrial electron transport remains essential (through the action of ribonucleotide reductase) in generating deoxyribonucleiotides from nucleotides as well producing ROS in collusion with oncogenes [83] and molecular oxygen [84] promoting genetic damage in normal cells and therapy resistance in cancerous cells [85].Malignant cells also use reduced glutathione [81] or NADPH to combat oxidative stress and to support the oxidation of fatty acids in detached cells [86]