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While trophic support might promote peripheral repair and reduce the drive for central gain, it is unclear whether it can actively reverse entrenched pathological neural circuits or if it merely prevents further deterioration

The problem arises when this antioxidant defense is overwhelmed or depleted: *Excess HO damages thyroid follicular cells, triggering inflammation and immune activation *Oxidized thyroid proteins become neoantigens - foreign-looking molecules that the immune system attacks, initiating or amplifying autoimmune thyroid disease *Lipid peroxidation in thyroid cell membranes disrupts hormone synthesis and cellular integrity *Chronic oxidative stress accelerates thyroid cell apoptosis and fibrosis - the hallmarks of Hashimoto's progression A landmark review published in Antioxidants (MDPI, 2021) - "The Influence of Oxidative Stress on Thyroid Diseases" - confirmed that decreased glutathione levels appear to be a distinctive parameter related to the activation and development of oxidative stress in Hashimoto's thyroiditis, and that antioxidant interventions represent a promising therapeutic avenue for autoimmune thyroid conditions

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612 % des Ausgangsgewichtes erreicht und mit Semaglutid eine Gewichtsabnahme um circa 15 % (Grafik 2) (49, 50, 51, 52, 53)