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What these mechanisms do NOT prove: that these pathways are activated to the same degree, duration, or tissue specificity in a human taking an oral capsule with unknown systemic bioavailability

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However, sulforaphane co-administration blunts ROS elevation through increased expression of the antioxidant scavenger, glutathione, thereby preventing neuronal cell death ( Given the central role of oxidative stress in seizure-related neuronal damage, mitochondria emerge as a potential target for oxidative insult

DHODH and cancer: promising prospects to be explored