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Peroxiredoxin II is essential for preventing hemolytic anemia from oxidative stress through maintaining hemoglobin stability

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Moreover, an indirect effect of testosterone through the activation of GH/IGF-1 has been shown by the evidence that pretreatment with testosterone (or estradiol in female) increases GH response to stimuli

Notably, CAC defects often result in life-threatening conditions due to impaired mitochondrial -oxidation, leading to increased fatty acid accumulation in the cytoplasm