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Current evidence, including magnetic resonance imaging studies, suggests that skeletal muscle alterations resulting from GLP-1 RA-induced weight loss, such as enhanced insulin sensitivity and reduced muscle fat infiltration, are adaptive, thereby improving muscle quality despite concurrent reductions in lean mass [112]

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Although quinone metabolites typically carry the risk of hepatotoxicity, previous studies have found that glucuronidation competes with this bioactivation pathway, forming a detoxification mechanism, which may be an important reason for its clinical safety profile (7,139)

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