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Interestingly, the PPAR-regulated gene CD36 was significantly upregulated only in eWAT from P5-treated DIO mice (Fig

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Emerging evidence suggests that intrinsic beta-cell factors, including oxidative stress and post-translational modifications (PTM) of beta-cell antigens, may also contribute to their immunogenicity, shedding new light on the multifaceted pathogenesis of T1D

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