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The iron released during this process is exported via ferroprotein ( FNP1 ) whose expression is modulated by heme and iron levels [18]

In saline-treated rats, Dox exposure caused sarcolemmal vacuolization, myofibrillar fragmentation, and extensive mitochondrial injury, including swelling, cristae disruption, and membrane dilation

Those that are targeted by the mediators IL-8 and growth-related oncoprotein-a (GRO-alpha) are ideal targets such as CXCR1, linked to cell activation and degranulation, and the high-affinity receptor (CXCR2), important in the chemotactic response