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Morita K, He S, Nowak RP, Wang J, Zimmerman MW, Fu C, et al

Chronic sleep deprivation or poor sleep quality impairs these restorative processes, potentially reducing glutathione synthesis capacity and increasing oxidative stress through various mechanisms including increased inflammatory signaling, impaired glucose metabolism, disrupted circadian rhythms affecting antioxidant enzyme expression, and increased psychological stress responses

doi: 10.1083/jcb.125.4.929

In previous I/R models, these effects include attenuation of oxidative stress, preservation of endothelial and microvascular integrity, modulation of nitric oxide signaling, suppression of inflammatory cytokine expression, and regulation of apoptotic pathways.55,62,63 These properties suggest that BPC 157 may represent a potential therapeutic candidate for I/R injury