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It counteracts the consequences of traumatic brain injury directly, reducing brain edema, hemorrhagic lacerations, and subarachnoid bleeding in experimental models

bioRxiv , 2025-06

In Long COVID, oxidative stress may be driven by: persistent immune activation mitochondrial dysfunction mast cell activation (MCAS) chronic inflammation autonomic nervous system dysregulation psychological stress (PTSD, illness trauma) Even everyday factors can worsen the burden: travel overexertion poor sleep emotional stress Supporting the Body Against Oxidative Stress While research is ongoing, strategies that may help reduce oxidative stress include: Foundational Support Anti-inflammatory nutrition Adequate hydration Sleep optimization Stress reduction Nervous System Regulation Breathwork Pacing strategies Reducing overstimulation Nutritional & Clinical Considerations Antioxidant support (as clinically appropriate) Mitochondrial support Addressing histamine and MCAS if present Always consult with a qualified provider before starting new treatments or supplements

For example, in the human brain, females exhibited increased binding of the CB 1 R with age, whereas males did not show any change (Van Laere et al., 2008)