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Unpublished results from the first phase II trial with the triple combination of semaglutide, cilofexor and/or firsocostat treatment for 24 weeks in 108 patients with NASH showed significant improvements in hepatic steatosis (measured with MRI) and liver injury (measured by serum alanine aminotransferase) in the combination treatment arm vs semaglutide monotherapy (56)

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With a growing body of evidence supporting the involvement of ferroptosis in autoimmune diseases, it is apparent that this cellular demise pathway is ubiquitously present and pivotal in the pathogenesis of autoimmune disorders [13, 14]

The anecdotal data in the real world is so clear it goes without saying