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Knockdown or pharmacological inhibition of components within this axis effectively reversed the ferroptosis-enhancing impact of butyrate, thus establishing a functional and causal relationship between mitophagy suppression and heightened Erastin responsiveness

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One of the hallmarks of mitochondrial dysfunction in ASD is when the disease is associated with inflammatory events, gastrointestinal issues, seizures, motor delays, fatigue, and lethargy [30]