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(2019) established that AKT activation by phosphatidylinositol 3,4,5-trisphosphate 3-phosphatase and dual-specificity protein phosphatase (PTEN) deficiency, a direct PI3K antagonist, maintained a refractory state of cells to treatment with cytarabine (Ara-C) and IDA through the improvement of glycolysis in the myeloid lines HL60 and KG1

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Therefore, one should be very careful in formulating hypotheses and pay attention to whether the results obtained in the experiments come directly from ascorbate or possibly from reactive oxygen species generated during cell supplementation