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The LD50 of GHK-Cu in animal models is many times higher than standard research doses, indicating a large therapeutic window

With its targeted mechanism of stimulating GH release through the ghrelin receptor without excessive cortisol or prolactin spikes, it offers a safer and more tolerable profile than older compounds like GHRP-6 or GHRP-2

These effects could lead to suppressing cyclin D1, re-expressing silenced tumour suppressor genes, inhibiting inflammation mediated by NF-B, or downregulating metastatic markers like MMP-2 and MMP-9