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The most extensively studied mechanism involves upregulation of vascular endothelial growth factor (VEGF), which promotes angiogenesis, the formation of new blood vessels at injury sites, providing the vascular supply necessary for accelerated tissue repair

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As noted by Manasyan et al., while these agents reduce insulin requirements and maintain adequate glucose control with fewer hypoglycemic events, they may simultaneously impede adequate nutrition as a result of delayed gastric emptying and present a risk of aspiration