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We utilized Students t-tests and Pearson correlations for parameter comparisons
Because GSH is a cofactor for GST isoforms, disruptions in GSH synthesis have the potential to severely disturb the detoxifying capacity of hepatic GSTs (Hardwick et al
Phase 4 (Post-Marketing): Monitors long-term safety, effectiveness, and rare side effects after the drug reaches the market
Although the comparison between 34 hr and 58 hr showed no differences in most metabolites except butyrates and OA, we also noticed that some metabolites peaked in 34 hr, while other metabolite kept increasing until 58 hr (cis-aconitate, succinate, nicotinamide (Fig
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