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[314] A moderate dose of caffeine enhances rapid eye movements' kinematics, such as saccade velocities and quick phases of optokinetic nystagmus, in unfatigued individuals, without affecting pursuit eye movements or visual perception, suggesting that caffeine's influence on oculomotor control may be mediated by changes in central catecholamines even in non-fatigued conditions
examined the expression of CPT-1 isoforms in photoreceptor cells and retinal pigment epithelial cells that are known to be affected morphologically and functionally in complete MTP deficiency and deficiency of long-chain 3-hydroxyacyl-CoA hydratase (LCHAD) [167]
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