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Groups 4 and 5 (semaglutide 1.0 mg and 2.4 mg, respectively) discontinued placebo infusions

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Evidence suggests that GLP-1 pathways influence the central nervous system via multiple routes: some agents and/or their downstream effects reach or affect the brain and bloodbrain barrier , altering glucose transport, microglial activation, and synaptic function in preclinical and early human studies

From a mechanistic perspective, GLP-1RAs like exenatide notably decreased the percentage of Th17 cells while promoting the proliferation of Tregs by influencing the signaling cascade encompassing phosphatidylinositol 3-kinase (PI3K), protein kinase B (AKT), along with forkhead box O1 (FoxO1) [147]