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Once in the nucleus, Nrf2 binds to antioxidant response elements (AREs) and induces the expression of over 500 cytoprotective genes, including superoxide dismutase (SOD) and glutathione peroxidase (GPx) 9,10,11 , thereby enhancing the cells resilience against more significant stressors

When laser light irradiates accumulated PS reagents (e.g., bilirubin), photoactivation generates toxic ROS and induces cell death to provide local antitumor therapy

Both collagen and glutathione are generally very well-tolerated since they are substances your body already produces

Inhibition of LILRB2 also plays a critical role in reprogramming the tumor-associated macrophage population toward a more active, inflammatory phenotype, which can further disrupt the immunosuppressive environment [126]