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Marx N, Husain M, Lehrke M, Verma S, Sattar N

In silico studies predicted that HCQ and other AMDs interacted with cGAS-DNA complex at a site necessary for binding to cGAS and its activation by DNA, and in vitro experiments confirmed that quinacrine blocked the binding between dsDNA and cGAS (78)

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Among these, TNF- is highlighted as a critical driver, with elevated levels in serum and peritoneal fluid of patients with active lesions, and its altered balance with Th1/Th2 cytokines linked to embryo toxicity and endometrial dysfunction (25)