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doi: 10.3389/fimmu.2025.1714608 86 AgustAMelnEDeMeoDLBreyer-KohansalRFanerR

Acknowledgements Supported by the Consejo Nacional de Ciencia y Tecnologa (CONACyT): Grant SALUD-2002-C-01-7615/A-1, Fellowship register no

Avila-Carrasco L, Garca-Mayorga EA, Daz-Avila DL, Garza-Veloz I, Martinez-Fierro ML, Gonzlez-Mateo GT

Although the direct cytotoxic effects of high cellular GSSG levels have been documented in a variety of cell types 33,34,35,36 , our cell culture studies with hepatocytes revealed that increased GSSG accumulation, despite being non-toxic per se, could sensitize hepatocytes to TNF-induced killing, suggesting that GSSG and TNF can synergistically induce hepatocyte death and that GSSG-triggered sensitization to TNF hepatotoxicity represents a clinically relevant process that may account for disease progression in NAFLD