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GA can enhance AMPK phosphorylation in a dose-dependent manner, inhibit the transcriptional activity of SREBP-1 and its downstream fatty acid synthase (FASN) and ACC to downregulate lipogenesis, thereby inhibiting hepatic lipogenesis and accumulation and reducing hepatic steatosis in fructose-induced NAFLD mice ( Previous studies on the mechanism of action of GA on NAFLD were based on pharmacological methods

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Clean handling, careful storage, and tidy reconstitution keep the process stable