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Loss of GSTM1 may therefore increase vulnerability not just by reducing detoxification capacity but also by weakening control of stress and cell death signalling cascades

Beyond its well-established role in glucose control in diabetes mellitus, GLP-1 has increasingly become the target of anti-inflammatory therapies for the management of conditions such as atherosclerosis, nonalcoholic steatohepatitis, nephropathy secondary to diabetes, and neurodegenerative disorders [44]

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