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16 Berenbaum hypothesized that the therapeutic potential of GLP-1 in OA may arise from targeting the crosstalk between macrophages and fibroblasts, which are known to drive inflammation, fibrosis, and downstream cartilage damage

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Its influence on intracellular repair signaling networks has led to growing research interest within experimental sports science and orthopedic models focused on tissue adaptation and recovery (4,5)