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Pulmonary vascular remodeling, a hallmark of PAH, is primarily driven by the excessive proliferation and reduced cell death of pulmonary artery smooth muscle cells (PASMCs) [140, 141]

We also introduce the protective effects and mechanisms of the associated agonists and inhibitors in various kidney diseases, which may provide valuable insights into novel treatment strategies for renal pathologies

In depressed patients, the expression and availability of glucocorticoid receptors are decreased, which prevents the HPA axis from exerting anti-inflammatory effects and promotes persistent inflammation (94, 95)

For example, recent findings indicate that HSPA1B facilitates the exosomal secretion of metalloproteinases from macrophages, thereby helping to mitigate fibrosis (Xiao et al., 2025)