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Furthermore, dual-action compounds engaging GLP-1 combined with glucose-dependent insulinotropic polypeptide (GIP) or glucagon pathways, alongside triple agonists incorporating all three receptors activation, have exhibited enhanced therapeutic potential

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Beyond BED, bulimia nervosa (BN) also shares core features of impulsive, dysregulated eating and recurrent binge episodes, and GLP-1related satiety and reward pathways are likely relevant in both conditions [21]

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