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Key liver function markers, including ALT, TBIL, and ALB, showed notable enhancements, with ALT levels decreasing from 348.96 U/L to 31.11 U/L in the observation group, compared to 347.90 U/L to 56.90 U/L in the control group

In A-induced AD model mice and APP23/PS45 double transgenic AD model mice, UA treatment promotes A clearance and improves cognitive deficits by restoring TFEB-dependent autophagy [217](Fig

TMAVA, a metabolite of intestinal microbes, is increased in plasma from patients with liver steatosis, inhibits -Butyrobetaine hydroxylase, and exacerbates fatty liver in mice

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