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Silencing TXNIP ameliorates high uric acid-induced insulin resistance via the IRS2/AKT and Nrf2/HO-1 pathways in macrophages

Your body is constantly exposed to things it needs to eliminate: metabolic byproducts, environmental chemicals, old hormones, even excess neurotransmitters

Collectively, the structural plasticity and autoinhibitory-loop control of the p300/CBP HAT core explain their capacity to catalyze L-lactylation in defined contexts