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This review discusses the molecular mechanisms of EGCGs anti-oxidative stress and chronic neuroinflammation, emphasizing its effects on autoimmune responses, neuroimmune system interactions, and focusing on the related effects on AD and PD

Preclinical studies have demonstrated that GLP-1 RAs attenuate alcohol intake and reduce the ethanol-induced activation of mesolimbic dopaminergic pathways, highlighting their ability to modulate dysregulated reward circuitry associated with AUD [6,21]

A similar effect of ascorbate in the case of CAT activity is also observed in rapidly proliferating mammalian cells, especially cancer cells, e.g., gastric cancer cells, glioblastoma, and carcinoma cells [74,75]

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