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Huperzine A: A promising anticonvulsant, disease modifying, and memory enhancing treatment option in Alzheimers disease

Patients with GSTM1-null genotype or chronic inflammatory conditions experience faster glutathione depletion during supplementation gaps

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RNA sequencing and pathway enrichment analysis show FOXO4-DRI significantly downregulates extracellular matrix-receptor interaction pathways, including fibronectin (Fn1), tenascin C (Tnc), and thrombospondin 2 (Thbs2), all critical components of fibrotic tissue architecture