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Metabolites from ellagitannins (e.g., urolithin A) and flavonoids block TLR4-MyD88-IKK signaling, blocking IB degradation and limiting NF-B nuclear translocation
when either of these is added to melanocytes or melanoma cell lines, melanogenesis is shifted toward pheomelanin formation
This issue is especially important for therapies directed at mitochondriaorganelle crosstalk, because the balance between reversible stress adaptation and irreversible bioenergetic collapse is strongly time-dependent, and the therapeutic window in patients is often substantially later than in experimental studies
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