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In another study, liraglutide was shown to prevent the onset of endoplasmic reticulum stress in HUVECs exposed to high glucose via dose dependent induction of mitochondrial fusion marker, OPA1, thereby inhibiting mitochondrial fragmentation and apoptosis [104]

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71 , 72 The exception to this occurs with Class I G6PD deficiency, where some affected individuals may have neutrophil dysfunction and increased susceptibility to infection