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A study using antibiotic treatment to deplete butyrate-producing microbes revealed that this type of dysbiosis supports Enterobacteriaceae expansion and inflammation through suppression of the peroxisome proliferator-activated receptor gamma (PPAR-) homeostatic signaling pathway [38]

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Spectroscopic and kinetic evidence for the thiolate anion of glutathione at the active site of glutathione S-transferase

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