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Prescription GLP-1 medications mimic the bodys own GLP-1 hormone, directly activating GLP-1 receptors to produce clinically significant effects, such as appetite suppression, improved insulin response, and weight loss

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This impairs triglyceride mobilization by interfering with PNPLA3 ubiquitylation and proteasomal degradation [51] , may suppress adipose triglyceride lipase activity to reduce hepatic triglyceride hydrolysis [52,53] , and can activate proinflammatory pathways such as STAT1 and NF-B [54,55]