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While direct evidence in human tinnitus tissue is scarce and no study has yet demonstrated HDAC isoform alterations specifically in the AC in tinnitus models, pharmacological inhibition of HDAC3 in the AC has been shown to accelerate learning and strengthen cortical encoding of behaviorally relevant sounds, demonstrating that HDAC-mediated mechanisms can directly regulate AC plasticity (Ateyakar and Bieszczad, 2026)

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NAD plays a critical role in cellular repair, energy production, and neurological function, which may help reduce fatigue, brain fog, and age-related decline